In addition, a recent study highlights the translational potential of lncRNA H19 by showing that the expression of this gene is suppressed in response to afterload augmentation in a pig model of left ventricular hypertrophy, in human cardiac tissues from patients with heart disease, in human stem cell-derived cardiomyocytes, and in sentient heart tissue [50,51]. The gene discussed is H19; the disease is left ventricular hypertrophy.