These inflammatory responses and the resulting tumor development were similarly observed when p120-catenin was knocked out in the intestine, squamous oral cavity, esophagus, forestomach, and a TP53 null noninvasive breast cancer model (Smalley-Freed et al., 2010; Smalley-Freed et al., 2011; Stairs et al., 2011; Schackmann et al., 2013). This evidence concerns the gene CTNND1 and neoplasm.