Ritter and Korber [41] demonstrated the key role of MIG in the self-healing of localized cutaneous leishmaniasis, confirming that lesions exhibited a strong expression of Th1-associated chemokines, such as CCL2/MCP-1, CXCL9/MIG, and CXCL10/IP-10. This evidence concerns the gene CXCL9 and cutaneous leishmaniasis.