LBR and attention deficit-hyperactivity disorder: In the depleted sera of the ADHD patient group, we found a decrease in α2-3 sialylation (interactions with MAL-II), a decrease in mannose structures (interactions with ConA), a decrease in di-/triantennary complex type N-glycans with bisecting GlcNAc (interactions with PHA-E), and an increase in Gal/GalNAc structures (interactions with RCA).