GRIN2A and schizophrenia: Both D2−like dopaminergic receptors and the subunit 2A of the N−methyl−D−aspartate receptor (NMDA) glutamatergic receptors (GRIN2A) are trafficked after internalization via lysosomal−mediated degradation, suggesting that a reduction in DYS levels may selectively alter the dopaminergic and glutamatergic pathways, which, in turn, play a crucial role in the pathobiology of schizophrenia [24,25,26,27,28,29].