Previously, we showed that in stimulated human primary T cells, UBASH3A and IL2 mRNA levels were inversely correlated [16], two T1D risk genetic variants in UBASH3A led to more UBASH3A transcripts and less IL2 transcripts [16], and a T1D-protective variant in UBASH3A led to less UBASH3A transcripts and more IL2 transcripts [9]. The gene discussed is UBASH3A; the disease is type 1 diabetes mellitus.