IL1B and COVID-19: Accumulating evidence has indicated that infection with SARS-CoV-2 leads to increased levels of proinflammatory cytokines such as IL-6, IL-1β, TNF-α, and IFN-γ, known as molecules “at the intersection of COVID-19 and cancer” [54] and also known to be involved in formation of fibrous tissue, thus creating an oncogenic and profibrotic inflammatory microenvironment.