GPR19 and Fanconi anemia: Using pathway analysis of this signaling network, it was evident that this small 32 DEP cohort was able to encapsulate an effective microcosm of the overall GPR19 perturbagen phenotype, i.e., the most enriched pathways included those linked to oncology (“pathways in cancer”, “breast cancer”), cell stress and fate (“cellular senescence”, “apoptosis”), DNA damage management (“non-homologous end-joining”, “Fanconi anemia pathway”), and longevity-associated factors (“FoxO signaling pathway”).