As opposed to other autoimmune kidney diseases such as MPA, GPA, and LN, variations in the copy number of FCGR3A (genes associated with activating FC γ receptors) rather than FCGR2B (associated with inhibition of FC γ receptors) were associated with increased disease risk in anti-GBM patients [40]. The gene discussed is FCGR3A; the disease is glioblastoma.