It is interesting to point out that three miRNAs (miR-146, which has an anti-inflammatory NFκB-dependent function [58,81]; miR-155, which improves skin inflammation, decreasing CTLA-4 expression [54,80]; miR-223, which is expressed by neutrophils, monocytes and eosinophils and has a proinflammatory function [66,78,79]) are found in AD and ACD pathogenesis, suggesting that some of the underlying inflammatory mechanisms might be the same for both diseases. The gene discussed is NFKB1; the disease is Alzheimer disease.