The following evidence supports the above view: (I) diabetes is caused by FOXM1 mutation [29]; (II) immune cell recruitment regulated by FOXM1 can regulate wound healing in human diabetes [30]; (III) FOXM1 induces hepatocytes to cause inflammation, injury, and fibrosis [31]; (IV) FOXM1 plays a critical role in the induction of mitochondrial biogenesis for human memory-like CAR-T cells from stem cells [32]; and (V) FOXM1 is involved in disease development through the regulation of disease-induced miRNAs [33]. Here, FOXM1 is linked to diabetes mellitus.