A study by Singh et al. (2021) showed that these cells were enriched in L. major infection and colonized skin with Staphylococcus epidermidis, leading to augmented skin inflammation in cutaneous leishmaniasis, without affecting type 1 immune responses [83], which is also observed in the generation of IL-17-producing T cells [84]. The gene discussed is IL17A; the disease is cutaneous leishmaniasis.