Previous studies have theorized that hsa-miR-146a is overexpressed in many viral diseases, targeting and inhibiting specific genes, such as STAT1, SOCS1/STAT3, TCL1, CXCL4, and NFκB in hepatitis B and C, EBV, human immunodeficiency type-1, and human T-lymphotropic type 1, respectively [43,44,45,46,47]. This evidence concerns the gene NFKB1 and viral load.