We corroborate that in the AA patients with collapsing FSGS, the percentage of patients with COVID-19 and high-risk alleles is more than 90%.1,2,9 On comparison, among the 11 non-AA patients with collapsing FSGS with known APOL1 status, we found three of four (75%) with high-risk variants (Figure 2). This evidence concerns the gene APOL1 and COVID-19.