FADS1 and gastric cancer: During the generation of substrates, a study revealed that overexpression of elongation of very long‐chain fatty acid protein 5 (ELOVL5) and fatty acid desaturases 1 (FADS1) in mesenchymal‐type gastric cancer cells (GCs), which promote the biosynthesis of PUFAs, was downregulated in intestinal‐type GCs due to the methylation of the ELOVL5 and FADS1 promotors.102