In pancreatic ductal adenocarcinoma, cancer cells undergoing ferroptosis released a type of damage‐associated molecular pattern named KRAG12D to tumor microenvironment, which is subsequently internalized by macrophages via AGER pathway, inducing a shift in macrophages towards a pro‐tumor M2‐like phenotype and tumor growth.197. The gene discussed is AGER; the disease is neoplasm.