During the last years, myelin oligodendrocyte glycoprotein (MOG) IgG antibody associated disorders (MOGAD), a newly defined entity of acquired demyelinating syndromes, has been recognized as separate disease entity with recently published diagnostic guidelines.1 Children and adults with MOGAD present with diverse clinical phenotypes such as monophasic or recurrent presentations of acute disseminated encephalomyelitis (ADEM), optic neuritis, or transverse myelitis, and less commonly with cerebral cortical encephalitis, brainstem–or sole cerebellar presentations. Here, MOG is linked to optic neuritis.