FAM3C and psoriasis: Our studies showed that both, TPA and IMQ, two inflammatory agents with very different mechanisms of action (Stanley et al, 1991; Van der Fits et al, 2009) upregulated ILEI protein levels and caused a more severe psoriasiform phenotype in K5‐ILEIind mice, indicating that ILEI overexpression could be considered as a psoriasis risk factor model that can be investigated in diverse psoriasis driver backgrounds.