Counteraction of the negative effects of JA-Ile and coronatine on SA signaling. The JA-precursor OPDA accumulates in Hx-treated plants upon infection. Accumulation of transcripts, such as LoxD and OPR3, which are involved in OPDA- and JA biosynthesis. Potentiation of the expression of SA signaling genes, such as PR1 and PR5, upon pathogen infection, while the expression of ABA- and ET-related genes is not affected. Inhibition of stomatal re-opening mediated by coronatine, thereby probably preventing the entry of the bacteria to the plant mesophyll. This evidence concerns the gene ERG and infection.