KEGG enrichment analysis showed that cytokine-cytokine receptor interaction, JAK-STAT signaling pathway, viral protein interaction with cytokine and cytokine receptor, rheumatoid arthritis, and autoimmune thyroid disease were the most abundant pathways, and studies have confirmed that most pathways are involved in the pathogenesis of GBS30–33. The gene discussed is SOAT1; the disease is rheumatoid arthritis.