Noteworthy, the complement cascade, which is part of the innate immune system involved in alcoholic liver disease [62], was enriched for the SE events in ELOVL7, LINC00665, NSUN4, and SRRM2. We also found that epithelial-mesenchymal transition (EMT) was enriched for the SE events in ELOVL7, SRRM2 and TBC1D5 and was depleted for DRC1 and LINC00665 in GSEA. The gene discussed is DRC1; the disease is alcoholic liver diseases.