There was an association of both EGPA subgroups (ANCA + and ANCA –) with variants at the TSLP, BCL2L11, and CDK6 loci and suggestive evidence for BACH2, Chromosome 10, and lipoma preferred partner (LPP), indicating that EGPA is characterized by certain genetic variants associated with the syndrome as a whole (56). This evidence concerns the gene BACH2 and eosinophilic granulomatosis with polyangiitis.