Next, we found that by using antibodies to HNRNPA2B1 in PCa cell lysates, pri-miR-93 was enriched (Figure 4E), while depleting METTL3 inhibited this interaction (Figures 4F and S2D, E), suggesting that the m6A placement on pri-miR-93 by the m6A reader METTL3 was necessary for the recognition of HNRNPA2B1. Here, HNRNPA2B1 is linked to posterior cortical atrophy.