ABL1 and gastritis: The src-and C-Abl (non-receptor tyrosine kinases)-mediated phosphorylation of CagA is necessary for an H. pylori-mediated cell infection (Poppe et al., 2007; Mueller et al., 2012), patients with H. pylori-associated gastritis had significantly higher levels of C-Abl in the gastric epithelium and gland.