This is supported by our observations in transcriptomic datasets of SMARCA4/2-deficient BIN-67 (SCCOHT)42 and A427 (NSCLC)27,43 cells ± restoration of SMARCA4 or SMARCA2 and in SMARCA4-deficient H1944 (NSCLC)44 cells ± SMARCA2 knockdown: SLC2A1 was the only gene related to glucose transport or metabolism that was consistently activated by SMARCA4/2 among these data sets (Supplementary Fig. 2e, f, Supplementary Data 4). This evidence concerns the gene SLC2A1 and non-small cell lung carcinoma.