Indeed, SMARCA4/2-deficient NSCLC cells expressed the lowest levels of GLUT1 while SMARCA4-deficient cells with intact SMARCA2 expressed intermediate levels of GLUT1 (Fig. 2c, right), suggesting the redundancy of SMARCA4/2 in promoting GLUT1 expression. This evidence concerns the gene SMARCA2 and non-small cell lung carcinoma.