SLC2A1 and non-small cell lung carcinoma: Validating this, re-expression of SMARCA4 or SMARCA2 in SCCOHT and NSCLC cells upregulated SLC2A1/GLUT1 expression (Fig. 2e, f, Supplementary Fig. 2g), accompanied with elevated glucose uptake (Supplementary Fig 2h), whereas ectopic expression of the ATPase dead mutant variant (K785R) of SMARCA4 failed to upregulate GLUT1 protein (Fig. 2g) and mRNA42 (Fig. 2h).