Accordingly, glucose uptake capacities of SMARCA4/2-deficient ovarian cancer cells were strongly reduced (Fig. 2b), consistent with their markedly low GLUT1 protein abundance compared to controls (Fig. 2c, left, Supplementary Fig. 2b); ectopic expression of GLUT1 in SMARCA4/2-deficient cells significantly increased glucose uptake (Supplementary Fig. 2c, d). This evidence concerns the gene SLC2A1 and ovarian carcinoma.