In a glioma cell line, overexpression of FABP7 led to protection against reactive oxygen species (ROS) stress via increased accumulation of lipid droplets (LDs), whereas knockdown of FABP7 in astrocytes resulted in lower LD formation and increased ROS-mediated apoptosis; a reduction in apoptotic cell death upon FABP7 overexpression was also noted (74). This evidence concerns the gene FABP7 and central nervous system cancer.