The oral squamous cell carcinoma (OSCC) cell line was cytotoxic after the treatment, and intracellular reactive oxygen species (ROS) buildup was generated. This therapy also triggered apoptosis-related morphological alterations and the extirpation of phosphatidylserine in OSCC cells. Protein and gene expression of cadherin-1 were also boosted by the treatments. The gene discussed is CDH1; the disease is oral cavity squamous cell carcinoma.