Finally, proteins correlated with negativity for ANA, oligoarticular course, and no iridocyclitis were involved in innate immune cell functions, actin cytoskeleton organization, and response to wounding, e.g., various regulators of glucose transport and metabolism previously associated with RA pathogenesis and regarded as putative therapeutic targets (144), SELP, involved in inflammatory cell recruitment into the RA joint (172–174), and CD226, associated with susceptibility to JIA and proposed as a candidate risk factor for the disease (175). The gene discussed is SELP; the disease is iridocyclitis.