In the cortex of ST-treated rats, we recorded a gradual decrease in NF-κB binding activity compared with the vehicle group (shamST), reaching 6 ± 7% and 3 ± 3% of control in the 16- and 24-h post-stroke groups (p = 0.0185 and p = 0.0098, respectively). This evidence concerns the gene NFKB1 and stroke disorder.