TNNI3K and familial dilated cardiomyopathy: Both variants showed strong genetic segregation with the phenotype consisting of DCM, CCD, and SVT, in line with the previously described pathogenic variants.1,2,4 In particular, the TNNI3K-p.His592Tyr variant (now reclassified as a class 4 likely pathogenic variant) was identified in 7 independent probands.