Considering that TBM shares similarities with HAND and ischemic stroke—the former also involves pathogen invasion and neuroinflammation, while the latter is a common complication of TBM—the findings of previous studies indicating that the inhibition of glutaminase is beneficial in these two disease models suggests a potential pathway for treating TBM (Table 2) [4, 107-109]. This evidence concerns the gene GLS and meningeal tuberculosis.