Methyltransferase-like 3 (METTL3) mediates the maturation and upregulation of miR-126-2p to activate the PI3K/Akt/mTOR pathway by directly binding to phosphatase and tensin homologues (PTEN) to promote ovarian cancer progression and tumorigenesis; moreover, knockdown of METTL3 inhibits ovarian tumour formation in vivo [22]. This evidence concerns the gene METTL3 and ovarian cancer.