Our study comparing patients with SARS due to COVID-19 against patients with SARS due to other causes suggests that the dysglycemia in patients with COVID-19 was not directly associated with the SARS-CoV-2 infection as previously proposed [27], but is due to greater insulin resistance, probably secondary to higher levels of counterregulatory hormones and cytokines and the use of medications, although beta cell dysfunction secondary to prolonged hyperglycemia, inflammation, and/or low beta-cell reserve cannot be ruled out [25]. This evidence concerns the gene INS and COVID-19.