alleviated neurological deficits and increased protein levels of postsynaptic density protein 95 (PSD-95), phosphorylated calmodulin-dependent protein kinase II (p-CaMKII), calmodulin, and N-methyl D-aspartate receptor subtype 2B (NR2B) while preventing ischemia-induced cellular damage. This evidence concerns the gene DLG4 and ischemia.