HDAC9 and cervical carcinoma: In 2012, the same study group published their second research article on this topic and indicated that HeLa cell treatment with BML-210 and retinoic acid downregulated HDAC classes I and II and determined changes in protein expression levels of dystrophin, neuronal nitric oxide synthase (NOS1), as well as different isoforms of dystrobrevin, hence inhibiting cell growth and inducing apoptosis in cervical carcinoma cells [118].