From a molecular point of view, endometriosis is characterized by molecular abnormalities such as a loss of AT Rich Interactive Domain 1A (ARID1A) function, Phosphatase and Tensin homolog (PTEN) inactivation, Phosphatidylinositol-4,5-Bisphosphate 3-Kinase Catalytic Subunit Alpha (PIK3CA), Catenin Beta 1 (CTNNB1) and Kirsten Rat Sarcoma Viral oncogene homolog (KRAS) activation [6]. Here, CTNNB1 is linked to endometriosis.