DMD and serum lipopolysaccharide activity: The increase in sarcolemma permeability found in vivo using Evans blue as a permeability tracer is similar to that previously observed in denervated skeletal myofibers [5] and in skeletal myofibers of mice suffering from endotoxemia or inflammation associated with a genetic defect in dystrophin [6,15], all corresponding to in vivo demonstrations of membrane permeabilization via Cx HCs.