This finding implies that IGF-1 might be considered an inflammatory mediator implicated in SARS-CoV-2-associated damage of the pulmonary gas exchange barrier, leading to hypoxia that would prompt ischemia or heart failure [43]; or even directly exerts its inflammatory impact on the heart muscle causing myocarditis demonstrated as elevated serum H-FABP levels. This evidence concerns the gene IGF1 and myocarditis.