In terms of the AML morphology, RUNX1-mutated were more likely to be myelomonocytic compared to non-mutated samples (FAB M4, 33 vs. 17%, p = 0.1), followed by AML with minimal differentiation and without maturation (FAB M0, 6 vs. 1%, p = 0.26; FAB M1, 22 vs. 17%, p = 0.29). The gene discussed is RUNX1; the disease is acute myeloid leukemia.