Steady transfection of SMARCC2 led to decrease in volume, weight, proliferative index, microvessel density and M2 macrophage accumulation of subcutaneous tumours produced by IMR‐32 cells in immune‐deficient mice, and these effects were eliminated by transfection of CNBP (Figures 7F and S8A,B). This evidence concerns the gene SMARCC2 and neoplasm.