A recent analysis proved that TNNI3K had direct interactions with three DCM‐linked sarcomeric proteins (viz, myosin‐binding protein C, troponin I and cardiac α‐actin); therefore, the loss of function of TNNI3K resulted in myocardial structural disorders and arrhythmias.98 The gene discussed is TNNI3K; the disease is cardiac arrhythmia.