have demonstrated that METTL3 promoted apoptosis, autophagy, and pyroptosis of glutamic acid-induced intrahepatic cholangiocarcinoma (ICC) by interacting with DGCR8 and modulating the miR-30b-5p/PIK3R2 axis in an m6A-dependent manner (30). This evidence concerns the gene DGCR8 and intrahepatic cholangiocarcinoma.