(18) found that miR-150 controls ATB function by inhibiting the expression of BCR signaling-associated genes including ELK1 (ETS transcription factor) (HGNC:3321), ETF1 (eukaryotic translation termination factor 1) (HGNC:3477), and MYB, and by changing the surface characteristics of the major histocompatibility complex II (MHC II), thereby regulating insulin resistance, obesity-induced inflammation, and the glucose tolerance of adipose tissue. Here, ETF1 is linked to obesity disorder.