FUBP1 and neoplasm: Similarly, it was reported that mutation of FUBP1 might lead to lysine‐specific demethylase 1+8a deficiency and contribute to the tumorigenesis of glioma, suggesting the tumor‐suppressive effects of FUBP1,46 whereas competitive binding to circACTN4 could activate MYC transcription and facilitate tumor progression in breast cancer.33