Though we show that IL-3-expressing TCRβ+ T cells are accumulated in RE from FITC-treated mice but not from untreated mice, thus suggesting that IL-3 at the challenge phase is likely responsible for its effect on basophil extravasation, it does not exclude a possible role of IL-3 during the sensitization phase of ACD. Here, IL3 is linked to granular corneal dystrophy type II.