Thus, Ly and colleagues (Ly et al., 2010) observed a positive correlation between cancer progression and the polarization of macrophages towards M2-like since the expression of the F4/80, CD11c, PPAR-g, and CD163 genes are similar to the levels found for tissue repair, excess angiogenesis, hormonal decompensation, and immunosuppression caused by the progression of tumor factors and the development of metastases (Yang et al., 2019), as shown in Table 1. The gene discussed is CD163; the disease is neoplasm.