In line with CD68+ myeloid population infiltration in the liver observed in the human CC sample, rodent samples and published data,12 we found gene expression for myeloid recruitment protein to be increased, Ccl2; in addition, markers of CD68+ myeloid population, F4/80 and Cd68, also increased during cancer‐cachexia development (FigureS2A–C). The gene discussed is CCL2; the disease is Cachexia.