The majority of the pathways in cancer hallmarks were enriched in steatotic liver grafts, for example, glycolysis, IL-2-STAT5, inflammatory response, complement, and reactive oxygen species, which not only indicated the strong link between steatotic liver graft injury and cancer development, but also suggested the importance of both metabolism and immune response in steatotic liver graft injury (Figure 8A). The gene discussed is IL2; the disease is cancer.