Previous studies reported that a modulator of p75NTR, LM11A-31, which can promote p75NTR-associated signaling through disinhibition and/or activation of the TrkA/BNTR-p75 complex, ameliorates DSD and DO in SCI mice and also blocks SCI-related urothelial damage and bladder-wall remodeling [44,45,46]. Here, NGFR is linked to disorder of sexual differentiation.