Glucose is a primary font of energy, and a hypothetical association between glucose metabolism and the effects of triorganotin derivatives was demonstrated in the human pluripotent embryonic carcinoma cell line NT2/D1 in which exposure to tributyltin inhibited glucose-6-phosphate and fructose-6-phosphate production via the inhibition of the transporter GLUT-1 [15]. Here, SLC2A1 is linked to embryonal carcinoma.