We also investigated the pathways for CEP55 molecular interactions and nominated its interaction with three proteins—KIF14, KIF23, and ECT2—as a potential pathway for tumor progression that can be targeted for therapeutic interventions where computational tools can also be employed through a cheminformatics approach to analyze the binding of several molecules to these targets, an approach that could be translated later to effective anticancer drugs. This evidence concerns the gene KIF14 and neoplasm.